![]() ![]() Recently, it was reported that the sodium (Na)/potassium (K) ratio was associated with blood pressure (BP) in a cross-sectional analysis. Therefore, measuring the urinary Na/K ratio in community settings is a potential population approach for counteracting hypertension. In conclusion, the association of the change in urinary Na/K ratio with hypertension and changes in systolic and diastolic BP can be explained by a change in alcohol intake, BMI, and urinary Na/K ratio. Moreover, the change in systolic BP and diastolic BP was positively associated with the change in urinary Na/K ratio. We assessed the relationship between change in urinary Na/K ratio and BP change using multiple regression analyses adjusted for age, sex, and change in body mass index (BMI) and alcohol intake. For each year, we compared the baseline characteristics according to the urinary Na/K ratio and BP level. Tome City introduced urinary Na/K ratio measurements during health checkups since 2017. We targeted 12,890 participants who attended the health checkup in Tome City, Miyagi between 20. Here, we assessed whether the urinary Na/K ratio change measured using the Na/K device was associated with BP change in a health checkup setting. A Na/K ratio self-monitoring device using spot urine was established recently. All rights reserved.Recently, the sodium (Na)/potassium (K) ratio was reported to be associated with blood pressure (BP). © American Journal of Hypertension, Ltd 2019. Kawasaki estimated sodium excretion Tanaka estimated sodium excretion blood pressure hypertension sodium excretion spot urinary sodium. The Kawasaki and Tanaka estimations of the 24-hour sodium excretion showed a much lower correlation than previously reported. Spot urinary measurements of sodium give a very poor understanding of the natriuresis occurring over the same 24-hour period. 24-hour and Kawasaki and Tanaka estimated potassium excretions correlated much better (r = 0.58, P < 0.0001). The 24-hour and spot urinary K/creatinine ratios correlated weakly (r = 0.47, P ≤ 0.0001). All Bland-Altman analyses showed poor agreement.The 24-hour and spot potassium concentrations correlated very poorly (r = 0.1158, P = ns). The 24-hour sodium excretion and the Kawasaki-derived spot urine sodium excretion correlated moderately (r = 0.3118, P = 0.0052). The 24-hour and spot FENa results showed a weak negative correlation (r = -0.222, P = ns). The 24-hour and spot Na/creatinine ratios correlated weakly (r = 0.2625, P = 0.0194). The 24-hour and spot Na concentrations correlated moderately (r = 0.4633, P < 0.0001). Our cohort was 58% male and the median age was 41 years. 77 had matched and complete 24-hour and spot urinary and serum biochemistry to examine.We compared the spot and 24-hour urinary sodium concentration, Na/Cr ratio, FENa, Kawasaki and Tanaka estimated sodium excretion as well as the potassium concentration, K/Cr ratio, Kawasaki and Tanaka potassium excretion. We examined 419 hypertensive patients from the UCL Complex Hypertension Clinic. We compared spot to matched 24-hour urinary electrolyte measurements. 24-hour urine collections are arduous to collect, so many centers use spot urinary measurements instead. Guyton's principals suggest that the 24-hour urinary sodium excretion reflects sodium ingestion over the same period. Sodium intake is correlated with the development of hypertension. ![]()
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